
Study with the several resources on Docsity
Earn points by helping other students or get them with a premium plan
Prepare for your exams
Study with the several resources on Docsity
Earn points to download
Earn points by helping other students or get them with a premium plan
Community
Ask the community for help and clear up your study doubts
Discover the best universities in your country according to Docsity users
Free resources
Download our free guides on studying techniques, anxiety management strategies, and thesis advice from Docsity tutors
Bell's palsy and stroke can both cause facial weakness or paralysis, making them difficult to distinguish initially. However, there are significant differences between the two conditions: Bell's Palsy Cause: Temporary facial nerve paralysis, often due to inflammation or viral infection. Symptoms: Facial drooping or weakness on one side Difficulty closing the eye on the affected side Loss of taste on the affected side of the tongue Increased sensitivity to sound Stroke Cause: Disruption of blood flow to the brain, often due to a blood clot or bleeding. Symptoms: Facial drooping or weakness on one side Slurred speech or difficulty understanding speech Weakness or numbness in one arm or leg, especially on one side Sudden severe headache Vision problems, such as double vision or blurred vision
Typology: Cheat Sheet
1 / 1
This page cannot be seen from the preview
Don't miss anything!
Abiesha Smith Neuroanatomy Wednesday, March 1st, 2023
Both Bell’s Palsy and a Stroke are medical conditions often accompanied by facial paralysis of varying degrees and regions. But whilst the two are non- mutually exclusive, they are not identical and cannot be substituted for the other. Bell’s Palsy, also know as Idiopathic Peripheral Facial Paralysis , is a deterioration of ipsilateral facial muscles that results from a disruption in function/injury of the facial nerve after said nerve exits the cerebral area known as the pons. In Bell’s Palsy, not only could the facial nerve become injured just after leaving the pons but could damage could also occur in branches of the facial nerves, resulting in a slew of varying physiological ramifications. If the muscles of the forehead (upper face) and the lower face —all of which are innervated by the facial nerve—are affected, full paralysis or weakness of that particular (ipsilateral) side of the facial muscles being innervated would occur (the characteristic droopy facial expression for which Bell’s Palsy is known): additional complications include smoothness in the forehead, retinal, and nasal regions of the face. A Stroke, on the other hand, is a disruption in function/deterioration of contralateral facial muscles that results from a lesion in the facial motor nucleus and damage to upper motor neurons. In a Stroke, when a lesion of upper motor neurons—originating from the right side of the cerebral cortex—occurs, the lower left side of the face is affected due to the contralateral synapsing of said upper motor neurons to the lower motor neurons responsible for innervation of facial muscles on the lower left side of the face. What separates a Stroke from Bell’s Palsy is a lack of physiological damage to the upper face in a Stroke: the forehead region, in a Stroke, is unaffected if the ipsilateral innervation of its muscles (from the ipsilateral cerebral cortex) is also unaffected by the lesion. As such, where ipsilateral damage to the upper and lower faces are characteristic of Bell’s Palsy—due to damage of any region of the facial nerve, its canals, and innervated lower motor neuron regions, thereof—a Stroke’s paralysis, be it upper or lower facial regions and left/right side of the face, will depend on the upper motor neurons being affected and whether said disruptions are either contralateral, ipsilateral, or both.