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EKG Rhythms: Questions and Answers for Medical Students, Exams of Health sciences

A comprehensive overview of ekg rhythms, including their characteristics, causes, and clinical significance. It features a series of questions and answers designed to test understanding of various ekg patterns and their interpretations. A wide range of rhythms, from normal sinus rhythm to ventricular fibrillation, and includes explanations of key concepts such as heart rate, rhythm, and axis deviation. It is a valuable resource for medical students and professionals seeking to enhance their knowledge of ekg interpretation.

Typology: Exams

2024/2025

Available from 10/31/2024

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EKG rhythms questions and answers
already passed
Normal Sinus Rhythm โœ”โœ”60-100 bpm
all complexes normal and evenly spaced (P, QRS, T)
Sinus Arrest โœ”โœ”- SA node doesn't fire
- notice absence of P-wave for a complete cycle (a missed cycle)
Sinus arrhythmia โœ”โœ”all complexes normal but rhythmically irreg
- normal finding (esp in young pts) that has to do with breathing (rate: inhale-increase, exhale-
decrease)
Sinus Bradycardia โœ”โœ”<60
normal sinus rhythm
Sinus Tachycardia โœ”โœ”>100 (100-150)
normal sinus rhythm
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pf4
pf5
pf8
pf9
pfa
pfd
pfe
pff

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EKG rhythms questions and answers

already passed

Normal Sinus Rhythm โœ”โœ” 60 - 100 bpm

all complexes normal and evenly spaced (P, QRS, T)

Sinus Arrest โœ”โœ”- SA node doesn't fire

  • notice absence of P-wave for a complete cycle (a missed cycle)

Sinus arrhythmia โœ”โœ”all complexes normal but rhythmically irreg

  • normal finding (esp in young pts) that has to do with breathing (rate: inhale-increase, exhale- decrease)

Sinus Bradycardia โœ”โœ”<

normal sinus rhythm

Sinus Tachycardia โœ”โœ”>100 (100-150)

normal sinus rhythm

Wandering atrial pacemaker โœ”โœ”Hint: try never to pick this

  • impulse originate from varying points in atria
  • variation in P wave contour, PR-I, PP-I and thus RR-I

P wave vs T wave โœ”โœ”P generally smaller than T

MAT (multifocal atrial tachy) โœ”โœ”- impulse originates at diff places in atria so P waves diff and

intervals might not be consistent

  • assoc w/ severe pulm dz

Atrial Fibrillation โœ”โœ”A: 350-450 (atria quivering)

  • irreg-irreg rhythm (R-RI=irreg)

unsure/no P-wave (non-distinguishable)

  • irreg rhythm BUT reg QRS!

Danger: increase the risk of thromboemoblic events don't convert unless occurring less than 48 hrs, if don't know pt need to be put on thrombolytics)

Atrial Flutter โœ”โœ”A: 250- 350

Premature junctional contractions (PJC) โœ”โœ”- premature slightly widened QRS

  • +/- inverted P', before or after QRS, sometimes disappears w/in QRS

Premature atrial contractions (PAC's) โœ”โœ”- originates suddenly in irritable atrial foci

  • P' is earlier than expected and diff shape than P (often have a pause following PAC)
  • can occur in Bigeminy, Trigeminy, Quadgeminy pattern

Supraventricular Tachycardia (SVT)

aka

Paroxysmal atrial tachycardia (PAT) โœ”โœ”150-250 "sudden rapid heart rate"

  • an irritable atrial focus discharging
  • very fast and EVEN!
  • +/- inverted P waves
  • P often overlaps prior T wave

First-degree AV block โœ”โœ”- PRI >5 boxes/.20 sec

  • Fixed but prolonged PRI

(consistent but long)

  • normally get bradycardia here

Second-degree block: Mobitz Type I Wenckebach) โœ”โœ”"walk it back"

  • PRI gradually lengthens then drops QRS "grouping and then a miss"
  • typically pattern exists

(constant P-P interval, QRS is what is moving back)

  • not really serious or dangerous

Second-degree AV block: Mobitz Type II โœ”โœ”- normal PRI then sudden drop of QRS

  • P wave doesn't always produce QRS
  • P-R interval is constant (diff from 3rd degree)
  • no hint just drops out -> is serious and dangerous pt needs tx!
  • tend to be every other, so drops HR by 1/2) --> def bradycardia (rate=40bpm)

Third-degree AV block (complete block) โœ”โœ”rate around 40's

  • no relationship b/t P waves and QRS complexes (QRS slower than P rate)
  • P-P reg (atrial reg) & R-R reg (vent reg)

but NOT connected (i.e P-R inconsistent)

  • no P waves (from vent foci)
  • Wide QRS

(serious, death like rhythm)

  • called "dying heart" rhythm...occasional ventric beat b4 death (asystole)

Accelerated idioventricular rhythm (AIVR) โœ”โœ”40-

  • occur in short burst, usually following MI
  • mostly asx with no progression to vtach / vfib

Ventricular tachycardia โœ”โœ”150-250 (>120 from onysko)

  • ventricle irritated and moving fast
  • rapid, bizarre, wide QRS complexes
  • 1 large QRS after another!

(in vtach pt may not have pulse)

Ventricular Flutter โœ”โœ”250-

  • smooth sine-waves w/ similar amp
  • can lead to deadly arryth

goes right into vfib

Tosades de Pointes โœ”โœ”Flutter 250-

  • type of vtach, can lead to vfib
  • ribbon like fashion (hallmark: up and downward deflection of QRS)
  • d.t hypomagnesium

Ventricular Fibrillation โœ”โœ”350-

  • "chaotic"
  • mult vent foci rapidly discharging -> erratic vent rhythm
  • no identifiable waves
  • RESPOND IMMEDIATELY!
  • no pulse or perfusion, pt=dead

R atrial hypertrophy โœ”โœ”tall P waves! (in lead II, III, and aVF)

  • 2.5 boxes

cause: pulm HTN, COPD, Pulm emboli

A block in the Bundle Branch produces a delay in depol of the ventricle that it supplies

(note: can't read ischemia b/c BBB distort this)

if have L & R BBB = complete block

R Bundle Branch Block โœ”โœ”- wide QRS >3 boxes (0.12)

  • V1/V2 "bunny ears" (2-R waves)

(V1/2/3 all up)

  • common, doesn't have much path assoc

L Bundle Branch Block โœ”โœ”- wide QRS >3 boxes (0.12)

  • V5/V6 "bunny ears" (2-R waves)

(V1/2/3 all down)

  • not as common, more patho

Myocardial Infarction (MI) โœ”โœ”Ischemia: inverted Twave

Injury: ST seg elevation

Necrosis: Q wave present

  • area of infarct doesn't conduct electrical activity
  • infarction=cell death

(A MI (heart attack is) when blood vessels that supply blood to the heart are blocked, preventing enough oxygen from getting to the heart. The heart muscle dies or becomes permanently damaged)

Ischemia โœ”โœ”- T wave inversion

  • Ischemia is caused by a decrease in oxygen to the myocardial tissue (hypoxia/diminished blood supply)
  • can still save heart cells/reverse

Injury โœ”โœ”ST segment elevation (a sign of acute injury going on presently) (look for sad face)

  • Injury indicates the acuteness of an infarct (acute or recent)
  • occlusion of circumflex artery

Posterior wall โœ”โœ”since no post lead look in V1 for unusually large R wave

  • occlusion of right coronary artery

Angina pectoris โœ”โœ”...

Unstable angina โœ”โœ”...

Digitalis Effect โœ”โœ”- shortened QT interval

  • characteristic down-sloping ST depression (SCOOPING ST seg)

Hypercalcemia โœ”โœ”Short/absent QT segment

(tooo healthy --> short QT (skinny))

Hypocalcemia โœ”โœ”(not healthy (not taking vit like Ca++) so look like a hipo which is large ->

long QT)

Hyperkalemia โœ”โœ”tall, peaked and narrow T

severe --> flattening of P wave, wide QRS, and tall T='sine wave'

Hypokalemia โœ”โœ”- flat T present

  • depressed ST seg
  • U wave to prominent U wave

Lown-Ganong-Levine Syndrome โœ”โœ”AV node by passed, so short PRI

P adjacent to QRS

Pacemakers โœ”โœ”pacemaker spike (may be small; sometimes missed)

  • not supraventricular so wide QRS

Pericarditis โœ”โœ”ST segment elevated in ALL leads

T wave: may be elevated off the baseline

  • Lup/Rdown = Left axis deviation
  • Rup/Ldown = Right axis deviation
  • Specific axis degrees: determine type of deviation, choose most iso-electric line and go to that line on the circle chart. Go 90 degrees into the good quadrant (the one you know you're in) and that will tell you the exact degrees.

Axis pic โœ”โœ”

Wide QRS โœ”โœ”Vtach, PVC, 3o AV block, BBB

Rate 40's โœ”โœ”3o AV block, 2o AV block type II, or ventricular rhythm