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A comprehensive overview of ekg rhythms, including their characteristics, causes, and clinical significance. It features a series of questions and answers designed to test understanding of various ekg patterns and their interpretations. A wide range of rhythms, from normal sinus rhythm to ventricular fibrillation, and includes explanations of key concepts such as heart rate, rhythm, and axis deviation. It is a valuable resource for medical students and professionals seeking to enhance their knowledge of ekg interpretation.
Typology: Exams
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Normal Sinus Rhythm โโ 60 - 100 bpm
all complexes normal and evenly spaced (P, QRS, T)
Sinus Arrest โโ- SA node doesn't fire
Sinus arrhythmia โโall complexes normal but rhythmically irreg
Sinus Bradycardia โโ<
normal sinus rhythm
Sinus Tachycardia โโ>100 (100-150)
normal sinus rhythm
Wandering atrial pacemaker โโHint: try never to pick this
P wave vs T wave โโP generally smaller than T
MAT (multifocal atrial tachy) โโ- impulse originates at diff places in atria so P waves diff and
intervals might not be consistent
Atrial Fibrillation โโA: 350-450 (atria quivering)
unsure/no P-wave (non-distinguishable)
Danger: increase the risk of thromboemoblic events don't convert unless occurring less than 48 hrs, if don't know pt need to be put on thrombolytics)
Atrial Flutter โโA: 250- 350
Premature junctional contractions (PJC) โโ- premature slightly widened QRS
Premature atrial contractions (PAC's) โโ- originates suddenly in irritable atrial foci
Supraventricular Tachycardia (SVT)
aka
Paroxysmal atrial tachycardia (PAT) โโ150-250 "sudden rapid heart rate"
First-degree AV block โโ- PRI >5 boxes/.20 sec
(consistent but long)
Second-degree block: Mobitz Type I Wenckebach) โโ"walk it back"
(constant P-P interval, QRS is what is moving back)
Second-degree AV block: Mobitz Type II โโ- normal PRI then sudden drop of QRS
Third-degree AV block (complete block) โโrate around 40's
but NOT connected (i.e P-R inconsistent)
(serious, death like rhythm)
Accelerated idioventricular rhythm (AIVR) โโ40-
Ventricular tachycardia โโ150-250 (>120 from onysko)
(in vtach pt may not have pulse)
Ventricular Flutter โโ250-
goes right into vfib
Tosades de Pointes โโFlutter 250-
Ventricular Fibrillation โโ350-
R atrial hypertrophy โโtall P waves! (in lead II, III, and aVF)
2.5 boxes
cause: pulm HTN, COPD, Pulm emboli
A block in the Bundle Branch produces a delay in depol of the ventricle that it supplies
(note: can't read ischemia b/c BBB distort this)
if have L & R BBB = complete block
R Bundle Branch Block โโ- wide QRS >3 boxes (0.12)
(V1/2/3 all up)
L Bundle Branch Block โโ- wide QRS >3 boxes (0.12)
(V1/2/3 all down)
Myocardial Infarction (MI) โโIschemia: inverted Twave
Injury: ST seg elevation
Necrosis: Q wave present
(A MI (heart attack is) when blood vessels that supply blood to the heart are blocked, preventing enough oxygen from getting to the heart. The heart muscle dies or becomes permanently damaged)
Ischemia โโ- T wave inversion
Injury โโST segment elevation (a sign of acute injury going on presently) (look for sad face)
Posterior wall โโsince no post lead look in V1 for unusually large R wave
Angina pectoris โโ...
Unstable angina โโ...
Digitalis Effect โโ- shortened QT interval
Hypercalcemia โโShort/absent QT segment
(tooo healthy --> short QT (skinny))
Hypocalcemia โโ(not healthy (not taking vit like Ca++) so look like a hipo which is large ->
long QT)
Hyperkalemia โโtall, peaked and narrow T
severe --> flattening of P wave, wide QRS, and tall T='sine wave'
Hypokalemia โโ- flat T present
Lown-Ganong-Levine Syndrome โโAV node by passed, so short PRI
P adjacent to QRS
Pacemakers โโpacemaker spike (may be small; sometimes missed)
Pericarditis โโST segment elevated in ALL leads
T wave: may be elevated off the baseline
Axis pic โโ
Wide QRS โโVtach, PVC, 3o AV block, BBB
Rate 40's โโ3o AV block, 2o AV block type II, or ventricular rhythm