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A comprehensive set of questions and answers related to the ibhre ceps exam, focusing on cardiac electrophysiology. It covers a wide range of topics, including arrhythmias, pacing, and electrophysiology procedures. Valuable for students and professionals preparing for the ibhre ceps exam.
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Type of pause seen after PVC - ANSWER Compensatory pause Type of pause seen after PAC - ANSWER Non-compensatory pause What would lead I look like in BiV pacing - ANSWER small or isoelectric because activating both V at the same time Inferior STEMI best seen in which leads - ANSWER inferiors Large (+) p wave in II - ANSWER Right Atrial enlargement (RAE) Notched P in II; biphasic late (-) p in V1 - ANSWER Left Atrial Enlargement (LAE) Elevated R in V1 - ANSWER Right Ventricle hypertrophy electrolyte imbalance associate with SQTS - ANSWER hypercalcemia electrolyte imbalance associate with LQTS - ANSWER hypocalcemia Which 2 leads are usually similar in configuration because they measure along the same horizontal axis - ANSWER I & v PVC location: (-) II, III, avF - ANSWER low, apex PVC location: (+) II, III, avF - ANSWER high, outflow tract PVC location: transition before v3 - ANSWER Left sided PVC location: transition after v3 - ANSWER Right sided Delta waves: (-) v1 - ANSWER Right sided Delta waves: (+) v1 - ANSWER Left sided Delta waves: (-) II, III, avF - ANSWER posterior Delta waves: (+) II, III, avF - ANSWER anterior Delta waves: (+) v2- 6 - ANSWER posterior septal
Delta waves: transition v1- 2 - ANSWER septal Indication for ablation of AP in pt w/ no SVT - ANSWER shortest pre-excited R-R <220 in AF increased risk of AP through pathway leading to VF Conduction through the AV node with delay or block, resulting in a broader QRS - ANSWER Aberrancy Electrical impulses trigger cardiac muscle contraction by controlling the flux of which ion across membrane - ANSWER Calcium Ventricle activation time measurement - ANSWER Beginning of Q to peak of R cSNRT= - ANSWER cSNRT=SNRT-SCL AVNRT response to adenosine - ANSWER SVT terminates with an A AVRT response to adenosine - ANSWER Ruled out, if SVT continues with AV block VA<AV - ANSWER AVRT Ashman's phenomenon - ANSWER aberration occurring when a short cycle follows a long one in AF RBBB morphology in v6 - ANSWER wide S wave LBBB morphology in v6 - ANSWER opposite of v Heart block type: A, H, no V - ANSWER 2nd type 2 - infranodal block Heart block that resolves during exercise is located in - ANSWER AVN Most rare hemiblock - ANSWER Left posterior Hemiblock Does antidromic AVRT have a delta wave? - ANSWER Yes Does orthodromic AVRT have a delta wave? - ANSWER No (AP conducts retrograde) When would you use precordial thump - ANSWER in pulseless VT when defib is not nearby
Oral drug most similar to Lidocaine - ANSWER Mexiletine Drug that decreases theophylline levels - ANSWER Mexiletine Most common toxic effect of antiarrhythmic drugs - ANSWER proarrhythmic 1st line class 1c drug on symptomatic AF pts w/ recent onset w/ no structural or ischemic heart disease - ANSWER Flecainide 1st line action for pt w/ narrow complex SVT - ANSWER Vagal maneuvers Side effects of amiodarone - ANSWER Acute - AV block, hypotension, bradycardia Chronic - pulmonary fibrosis, peripheral deposition leading to bluish discoloration, arrhythmias, hypo/hyperthyroidism, photosensitivity (corneal deposition) Drugs to avoid in WPW - ANSWER Any drug that blocks the AVN can increase the risk of rapid bypass Adenosine Beta Blockers Calcium Channel Blockers Digoxin Drugs that may be beneficial in WPW - ANSWER Flecainide & Procainamide Rhythm control for AF & AFL - ANSWER 1c & III Rate control for AF & AFL - ANSWER II & IV Cardiac Active Sympathetic catecholamines - ANSWER DINED Dopamine Isuprel Norepinephrine Epinephrine Dobutamine Beta Blocker Contraindications - ANSWER Asthma Heart block COPD Diabetes Extremities PVD Benefits of Vasopressin over Epinephrine in VF/pulseless VT - ANSWER Reduced cardiac ischemia & irritability one-time does to simplify administration
reduced propensity for VF ACE inhibitors reduce - ANSWER vasoconstriction & hypertension Medical therapies indicated for pt in anaphylactic shock - ANSWER IV fluid & epinephrine Cardiotonic drugs like Digitalis - ANSWER Slow HR & increase force of contraction Contrast is filtered out via - ANSWER Kidneys Deg and time of tilt table test - ANSWER 60-90 degrees 20 - 45 min SA node conduction time (SACT) - ANSWER SACT=(return interval-BCL)/ Echo beat - ANSWER impulse travels down to V via slow pathway & back up to A via fast pathway Tach happenings in DDD pacing when the paced V wave passes retrograde up to the AVN which is sensed as an A then the V is paced too soon - ANSWER PMT Type of AP that can conduct both ways - ANSWER manifest Type of AP that can only conduct retrograde - ANSWER Concealed Normal para-hisian response - ANSWER high output: A capture low output: V capture at longer time Epicardial punture location - ANSWER subxyphoid AV dissociation & H before each V - ANSWER BBRVT Criteria for BBRVT - ANSWER HIS precedes QRS HV 55- 160 if PPI-TCL<30 - ANSWER within circuit Phased Array - ANSWER ICE Morady Pacing - ANSWER VAAV=AT VAV=AVNRT PVC @ time of HIS (HIS is refractory), if impulse reaches A - ANSWER pathway
follows A pace, prevents inappropriate inhibition of the ventricular system by the atrial system - ANSWER Ventricular blanking period (VBP) follows v pace, prevents t wave sensing in ventricle - ANSWER Ventricular refractory period (VRP) Pacemaker therapy class indications - ANSWER 1: therapy good 2: conflicting 3: therapy not useful/harmful What class for ppm would a child with congenital heart disease: sinus node dysfunction w/ symptomatic bradycardia - ANSWER Class 1 What call for ppm would a teen w/ post op 3rd deg heart bloc w/ return of normal conduction in a few days - ANSWER Class 3 LBBB + RBBB - ANSWER Complete heart block Patients have LBBB & pacing LV before RV results in syncronization - ANSWER CRT Most common reason for lead removal - ANSWER infection Position 1 of pacer code - ANSWER Chamber that is paced O = none, A = atrium, V = ventricle, D = both A and V Position 2 of pacer code - ANSWER Chamber that is sensed O = none, A = atrium, V = ventricle, D = both A and V Position 3 of pacer code - ANSWER Mode of Response O, T (triggered), I (Inhibited), D Position 4 of pacer code - ANSWER Rate responsiveness: Indicates the programmability of the pacemaker Ability to adjust the HR based on physiologic need O = none, R = rate modulation Position 5 of pacer code - ANSWER Multisite pacing: Indicates that the pacer can pace multiple sites O = none, A = atrium, V = ventricle, D = dual Negative terminal of the pacer should be connected to - ANSWER distal electrode
During DDR pacer change out, new generator is attached to leads, but no pacing happens until generator is placed in pocket. Why? - ANSWER Unipolar leads: positive pole was not connected until pacer generator makes contact w/ tissue Minimum amount of mA required to elicit regular cardiac contractions - ANSWER Pacing threshold Wedensky hysteresis effect - ANSWER stimulation threshold may vary depending on whether turning up or down Rate responsive devices are beneficial in pts w/ - ANSWER SA node dysfunction Normal lead impedance - ANSWER 500 ohm Broken lead impedance - ANSWER 2500 ohm eroded insulation impedance - ANSWER 80 ohm When to not used synchronized cardioversion - ANSWER fast VT, Vflutter, VFib because no distinct QRS PTs w/ DDDR who exhibit retro VA conduction are prone to develop - ANSWER PMT Chronaxie= - ANSWER 2x rheobase is the minimum TIME to cause a contraction @ twice the Rheobase Class of drugs that can significantly raise stimulation threshold and cause LOC - ANSWER 1c Fastest rate that V can be paced in response to sensed A events - ANSWER Max tracking rate When is stimulation threshold usually lowest - ANSWER At time of implant What can reduce the acute rise in thresholds with in the first 2 months after initial ppm implant - ANSWER IV steroids or steroid eluting tip When is stimulation threshold the highest - ANSWER Acute rejection phase (1mo) High sensitivity = - ANSWER less sensitive Lower sensitivity = - ANSWER more sensitive
AFFIRM Study - ANSWER compare rate-control and rhythm-control strategies for the treatment of AF. AFFIRM demonstrated no survival advantage between rate- control (using ß-blocker, calcium channel blocker and/or digoxin) and rhythm-control strategies. What type of rate modulation requires a special lead - ANSWER temperature Effect of smaller electrode on pacing lead impedance - ANSWER increase Protects the pacemaker circuitry from cardioversion/defib energy circuits - ANSWER Zener Diode Plateau at which a normal amount of current is required to excite the tissue regardless of lengthening pulse width - ANSWER Rheobase Advantage of rate responsive or rate adaptive AV delay - ANSWER permits tracking at higher rates Most common cause of pacemaker failure to output - ANSWER oversensing To reduce incidence of safety pacing or functional non-sensing in pts w/ frequent ectopy - ANSWER shorten V blanking period Drug of choice in Afib conducting down bypass tracts - ANSWER Procainimide Drug class that decreases conduction velocity increases refractory period decreases automaticity - ANSWER 1a Class 1a may cause EADs or DADs - ANSWER EADs 1a drug not to use in MI or CHF - ANSWER Disopyramide 1a drug w/o many serious side effects (besides hypotension and QT lengthening) & not likely to increase digoxin levels - ANSWER Procainamide Drug class that suppresses DADs - ANSWER 1B Class that has little effect on conduction velocity decrease ERP decrease AP decrease automaticty - ANSWER 1b
Why 1b have little effect on atrial tissue - ANSWER AP in A is shorter than V 1b drug used in pts with MI w/ frequent PVCs - ANSWER Lidocaine Drug effective for VTs caused by digitalis toxicity - ANSWER Phenytoid Drug class that significantly slows conduction velocity by does not prolong AP or refractory periods - ANSWER 1c Effect of 1c on DFT - ANSWER increases Affects catecholamines, mainly affects SA & AV nodes, blocks epinephrine which slows HR - ANSWER Beta blockers Beta blockers effect on DFT - ANSWER reduces DFT Reduces VF after MI Avoid use in WPW Contraindicated in pts w/ asthma - ANSWER Beta blockers Only class III that only prolong AP (not ERP) - ANSWER Dofetilide Class III affects which phase of the AP - ANSWER prolongs phase 2 Class that increases ERP and prolong AP - ANSWER III Most effective drug for VF and unstable VT - ANSWER Amiodarone Preferred drug for AFib pts in renal failure - ANSWER amiodarone Drug used in cardioversion which may lower energy required and allow for rapid carioversion - ANSWER Ibutilide Slows HR and increases force of contraction in pts in heart failure - ANSWER Digoxin Effective on monomorphic VT involving abnormal purkinje and exercise VT - ANSWER Verapamil Caution of class IV drug in WPW - ANSWER can enhance antegrade conduction through AP Class that decreases automaticity, conduction, and refractory - ANSWER IV
ICD primary prevention - ANSWER prevention of SCD in pts who have not had VA or SCA but increased risk ICD secondary prevention - ANSWER 2nd chance- survivors of SCA/ Prior VA VANISH Study - ANSWER superiority of ablation over escalating medication therapy for pts w/ recurrent VT despite antiarrhythmic medication Most common cause of SCD in individuals under 40 - ANSWER HCM exertion related polymorphic (bi-directional) VT - ANSWER Catecholaminergic polymorphic VT Coved ST elevation in leads V1/2 - ANSWER Brugada Treatment of SQTS - ANSWER Quinidine & ICD Outflow tract more likely occurring in men - ANSWER LVOT Outflow tract more likely occurring in women - ANSWER RVOT
BBB and axis of Fasicular VT - ANSWER RBBB & superior axis retrograde leg in Fasicular VT - ANSWER Left posterior fascicle Drug choice in IST - ANSWER Ivabradine Charge (Q) formula - ANSWER Q=It Unit of absorbed dose - ANSWER RAD (gray) Unit of does equivalent or occupational exposure - ANSWER REM Normal function in dual chamber pacing. Occurs when sensed P rate is fast than the programed maximum tracking rate (or URL) - ANSWER Pacemaker wenkebach When might automatic mode switching fail - ANSWER A flutter When pacing mode switches from atrial tracking to non tracking mode upon tach detection - ANSWER automatic mode switching
SCD-HeFT trial - ANSWER * NYHA II/III, CM, EF 35%
Tachycardia type: digitalis toxic arrhythmias, Torsades, VTs that respond to Ca blocking agents - ANSWER Triggered Treatment is to reverse underlying metabolic abnormality - ANSWER Automaticity Afterdepolarizations of sufficient magnitude may engage the rapid sodium channels - ANSWER Triggered m and h gates - ANSWER control ionic fluxes of sodium across membrane Class 1 subset that decreases conductivity - ANSWER 1a or 1c Drug class that has little effect of AP; works by decreasing sympathetic tone - ANSWER beta blockers Drug class that decreases sympathetic input causing more parasympathetic influence - ANSWER 4 Drug classes that effects mostly SA and AV nodes - ANSWER II, IV, V Drug class that can convert unidirectional block into bidirectional by increasing the long refractory of the fast pathway - ANSWER Class I Typical lateral, inferior, and anterior leads monitored for an EP study - ANSWER I, II, V IEGM typically records which phase of the AP - ANSWER Phase 0 Programmed impulses allow you to assess - ANSWER Refractory periods Automaticity of a focus Characteristics of reentry circuits HRA catheter position - ANSWER High lateral wall near the junction of the superior vena cava The smallest possible time interval between 2 impulses that can be conducted through that tissue - ANSWER Functional refractory period Occurs during phase 3 of the AP before the cell is fully repolarized - ANSWER Effective refractory period Longest coupling interval where a premature impulse will fail to propagate through tissue - ANSWER Effective refractory period
Catecholamine dependent VT can be tested in the EP lab with the infusion of - ANSWER Sympathomimetic agents Reentry termination depends on - ANSWER - Refractoriness of the tissue between the catheter and circuit
Energy
2nd deg heart block type 2 - ANSWER Some electrical signals get to ventricles and some don't 3rd deg heart block - ANSWER Electrical signals do not go from Atria to Ventricles at all Signal Average ECG