Docsity
Docsity

Prepare for your exams
Prepare for your exams

Study with the several resources on Docsity


Earn points to download
Earn points to download

Earn points by helping other students or get them with a premium plan


Guidelines and tips
Guidelines and tips

NSG 211 Pathophysiology Exam 1 Study Guide: Key Concepts and Definitions, Exams of Nursing

This study guide for nsg 211 pathophysiology exam 1 provides a comprehensive overview of key concepts and definitions related to disease processes. It covers topics such as inflammation, immunity, and cancer, offering a valuable resource for students preparing for their exam. The guide includes definitions of important terms, explanations of different types of infections and immune responses, and a breakdown of the stages of cancer.

Typology: Exams

2024/2025

Available from 01/27/2025

bryanryan
bryanryan 🇺🇸

3.9

(8)

11K documents

1 / 7

Toggle sidebar

This page cannot be seen from the preview

Don't miss anything!

bg1
NSG 211 Pathophysiology Exam 1 Study Guide(Burns)
Questions With Complete Solutions
Idiopathic - unknown cause of disease
iatrogenic - caused by treatment
Acute Infection - comes on fast, resolves quickly
Chronic Infection - Lasts longer than 2 months
Nosocomial - Health care acquired
Communicable - spreads from host to host
Notifiable - STD's must be reported when diagnosed
Epidemiology - Study of communicable disease
Mortality - Death rate
Morbidity - decreased quality of life due to condition
Prevalence - Snapshot of a disease
Incidence - How many occurrences in a certain time period
Homeostasis - balance of physiological systems
Hypertrophy - Increase in cellular size (HTN, exercise)
pf3
pf4
pf5

Partial preview of the text

Download NSG 211 Pathophysiology Exam 1 Study Guide: Key Concepts and Definitions and more Exams Nursing in PDF only on Docsity!

NSG 211 Pathophysiology Exam 1 Study Guide(Burns) Questions With Complete Solutions Idiopathic - unknown cause of disease iatrogenic - caused by treatment Acute Infection - comes on fast, resolves quickly Chronic Infection - Lasts longer than 2 months Nosocomial - Health care acquired Communicable - spreads from host to host Notifiable - STD's must be reported when diagnosed Epidemiology - Study of communicable disease Mortality - Death rate Morbidity - decreased quality of life due to condition Prevalence - Snapshot of a disease Incidence - How many occurrences in a certain time period Homeostasis - balance of physiological systems Hypertrophy - Increase in cellular size (HTN, exercise)

Hyperplasia - Increase in cellular number (can predispose to dysplasia/anaplasia) Metaplasia - change from mature cell A to B (abnormal change in the nature of a tissue) Dysplasia - Varied nuclei and cell size/shape Atrophy - Decrease in cellular size Hypoxia - lack of oxygen to an area Ischemia - lack of blood flow/supply to the area (tissue perfusion) Necrosis - death of most cells in an organ or tissue due to disease, injury, or failure of blood supply (localized) Apoptosis - death of cells that occurs as a normal and controlled part of an organism's growth Gangrene - an area of necrotic tissue which has been infected by bacteria Serous Exudate - white/yellow fluid, thin, seen most often Fibrinous Exudate - sticky Purulent Exudate - dead white blood cells, indicates infection (puss)

Immunity Process - phagocytosis (innate) > APC > Antibody (immunoglobulin) Production (B) and Cell mediated (T) Macrophage - presents foreign material to B-cell (as APC) starts adaptive immunity T-cells - activated to attack cells showing foreign antigens T-Helper - (CD4) crucial in both specific and innate immunity IgG - small, most abundant. Crosses Placenta (second response) IgM - First Responder IgA - in secretions IgE - Mast cells, Histamine, Type 1 Hypersensitivity Primary Immunity - IgM Secondary Immunity - IgG Natural Active - Acquired through active illness (Chicken Pox) Artificial Active - Acquired through immune activation by vaccination Natural Passive - Breast Milk (colostrum)

Artificial Passive - subject given antibodies produced by another organism (antivenom) Type I Immune Response - Allergic Reaction (histamine released by IgE) Type II Immune Response - Cytotoxic Hypersensitivity Type III - Immune complex hypersensitivity (Ag-Ab immune) Type IV - Cell mediated or delayed (TB skin test) Pathophysiology of Anaphylactic Shock - Type I hypersensitivity, hypotension & Edema, increased capillary permeability Pathophysiology of Burns - Shock, hypotension, pain, infection Treatment of Burns - need protein to build collagen Superficial thickness - Does not blister (sunburn) Partial thickness - blisters full thickness - all the way through Dermis Local infection - inflammation, pain (stays in affected area) Systemic infection - generalized symptoms (everywhere) Gram - - make endotoxins within cell membrane (shock)

Eosinophils % - 2-4 % Monocytes % - 3-8% Basophils % - 0.5-1%