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NSG 430 – End-of-Term Final Exam Prep Guide., Exams of Nursing

NSG 430 – End-of-Term Final Exam Prep Guide.

Typology: Exams

2024/2025

Available from 07/02/2025

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NSG 430 – End-of-Term Final Exam
Prep Guide.
WHAT IS THE TREATMENT FOR PERIPHERAL EDEMA?
A. DIURETICS
B. EXOGENOUS ALBUMIN
C. HYPOVOLEMIC FLUID BOLUS
D. ENDOGENOUS RETICULOCYTES
<<<CORRECT ANSWERS >>>B
PERIPHERAL EDEMA IS CAUSED FROM AN INCREASE IN PORTACAVAL PRESSURE FROM
PORTAL HYPERTENSION. ALBUMIN IS A PROTEIN GENERALLY MADE IN THE LIVER, THAT
WILL WORK TO PREVENT FLUID FROM LEAKING OUT OF THE BLOOD VESSELS AND INTO
THE TISSUES.
WHAT IS A NURSING INTERVENTION FOR PATIENTS WITH ASCITIES? (SELECT ALL THAT
APPLY)
A. MEASURING THE ABDOMIN EVERY DAY
B. FLUID BOLUS TO REPLACE LOST FLUIDS
C. MEASURING THEIR CALCIUM LEVELS
D. DAILY WEIGHT
E. DIURETICS
<<<CORRECT ANSWERS >>>A, D, E
MANIFESTED BY ABDOMINAL DISTENSION AND WEIGHT GAIN.
YOU MAY SEE SKIN STRIAE WITH THESE PATIENT.
IF IT IS SEVERE THE UMBILICITUS MAY BE DISTENDED.
THESE PATIENTS CAN HAVE HYPOKALEMIA FROM HYPERALDOSTERONISM OR DIURETIC.
MONITOR PATIENTS POTASSIUM LEVELS.
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NSG 430 – End-of-Term Final Exam

Prep Guide.

WHAT IS THE TREATMENT FOR PERIPHERAL EDEMA?

A. DIURETICS

B. EXOGENOUS ALBUMIN

C. HYPOVOLEMIC FLUID BOLUS

D. ENDOGENOUS RETICULOCYTES

<<>>B

PERIPHERAL EDEMA IS CAUSED FROM AN INCREASE IN PORTACAVAL PRESSURE FROM

PORTAL HYPERTENSION. ALBUMIN IS A PROTEIN GENERALLY MADE IN THE LIVER, THAT

WILL WORK TO PREVENT FLUID FROM LEAKING OUT OF THE BLOOD VESSELS AND INTO

THE TISSUES.

WHAT IS A NURSING INTERVENTION FOR PATIENTS WITH ASCITIES? (SELECT ALL THAT

APPLY)

A. MEASURING THE ABDOMIN EVERY DAY

B. FLUID BOLUS TO REPLACE LOST FLUIDS

C. MEASURING THEIR CALCIUM LEVELS

D. DAILY WEIGHT

E. DIURETICS

<<>>A, D, E

MANIFESTED BY ABDOMINAL DISTENSION AND WEIGHT GAIN.

YOU MAY SEE SKIN STRIAE WITH THESE PATIENT.

IF IT IS SEVERE THE UMBILICITUS MAY BE DISTENDED.

THESE PATIENTS CAN HAVE HYPOKALEMIA FROM HYPERALDOSTERONISM OR DIURETIC.

MONITOR PATIENTS POTASSIUM LEVELS.

WHAT ARE PATIENTS MORE AT RISK FOR GETTING?

A. SPONTANEOUS BACTERIAL PERITONITIS

B. URINARY TRACT INFECTIONS

C. STAPH INFECTIONS

D. MRSA

<<>>A

BECAUSE OF HOW CIRROHSIS CHANGES THE IMMUNE SYSTEM.

IT IS A BACTERIAL INFECTION FOUND IN THE ABDOMEN. BACTERIA NORMALLY FOUND IN

THE INTESTINES ESCAPES INTO THE PERITONEAL SPACE. MOST COMMON BACTERIA TO

CAUSE THIS IS E. COLI. -

NASTY

WHAT IS THE CAUSE FOR HEPATIC ENCEPHALOPATHY?

A. INCREASE BILIRUBIN LEVELS

B. HIGH A1C LEVELS

C. INCREASED ALBUMIN LEVELS

D. INCREASE IN AMMONIA LEVELS

<<>>D

IT CAUSES NEUROTOXIC EFFECTS: CHANGE IN MENTAL STAUS, CHANGE IN LOC. MAYBE BE

SUDDEN OR GRADUAL.

NORMALLY AMMONIA IS CONVERTED TO UREA IN THE LIVER AND EXCRETED. THE

DAMAGED LIVER DOESN'T DO THIS.

LACTULOSE IS GIVEN TO HELP TREAT THESE PATIENTS TO HELP THE BODY EXCRETE THE

AMMONIA.

A PATIENT IS SHOWING ASTERIXIS, WHICH INDICATES THAT AMMONIA IS CROSSING THE

BLOOD- BRAIN BARRIER. WHAT CAN THE NURSE DOCUMENT THIS SYMPTOM AS?

WHAT DRUG CAN WE GIVE TO PATIENTS TO DECREASE ESOPHAGEAL AND GASTRIC

VARICES?

A. NSAIDS

B. ANTICOAGULANTS

C. NONSELECTIVE B-BLOCKERS

D. CLOTTING FACTORS

<<>>C

THE GOAL IS TO PREVENT BLEEDING FROM DECREASING PORTAL HYPERTENSION. BETA

BLOCKERS WILL DECREASE PORTAL PRESSURE REDUCING THE RISK.

WHAT DRUGS MAY BE GIVEN FOR AN ACTIVELY BLEEDING ESOPHAGEAL OR GASTRIC

VARICES?

A. OCTREOTIDE

B. TXA

C. DESMOPRESSIN

D. VITAMIN K SUPPLEMENTS

<<>>A

THERE IS LESS SYMPTOMS ASSOCIATED WITH THIS DRUG. VASOPRESSIN MIGHT ALSO BE

GIVEN.

VITAMIN K WILL BE GIVEN AS A SUPPORTIVE MEASURE FOR AN ACUTE BLEED.

A BALLON TAMPANODE MIGHT BE USED AS MECHANICAL COMPRESSION OF A VARICE.

WHICH SHUNTING PROCEDURE IS A NONSURGICAL PROCEDURE DONE FOR 2 OR MORE

BLEEDING EPISODES FOR ESOPHAGEAL AND GASTRIC VARICES?

A. PORTACAVAL AND DISTAL SPLENORENAL SHUNT

B. TRANSJUGULAR INTRAHEPATIC PORTOSYSTEMIC SHUNT (TIPS)

C. REPEATED BAND LIGATION

D. PORTOSYSTEMIC SHUNTS

<<>>B

- TRANSJUGULAR INTRAHEPATIC PORTOSYSTEMIC SHUNT (TIPS) IS A NONSURGICAL

PROCEDURE DONE FOR TWO OR MORE MAJOR BLEEDING EPISODES. IT IS CREATED TO

REDIRECT PORTAL BLOOD FLOW, PLACED BEYWEEN THE SYSTEMIC AND PORTAL SYSTEMS.

- PORTACAVAL AND DISTAL SPLENORENAL SHUNT IS A SURGICAL PROCEDURE DONE FOR

TWO OR MORE MAJOR BLEEDING EPISODES.

- REPEATED BAND LIGATION AND PORTOSYSTEMIC SHUNTS ARE LONG TERM

MANAGEMENT METHODS.

WHAT IS A MAJOR COMPLICATION FROM CIRRHOSIS OF THE LIVER?

A. PORTAL HYPERTENSION

B. JAUNDICE

C. EDEMA IN THE HANDS AND FEET

D. CARDIOMEGALY

<<>>A- PORTAL HYPERTENSION

A PATIENT WITH PORTAL HYPERTENSION HAS SCLOROTIC VESSELS THAT IMPAIR BLOOD

FLOW CAUSING HYPERTENSION. THIS LEADS TO EVERYTHING TO BACK UP AND GET

ENGORGED/ ENLARGED.

WHAT ARE SOME COMPLICATIONS OF PORTAL HYPERTENSION? (SELECT ALL THAT APPLY)

A. SPLEENOMEGALY

B. ASCITES

C. GASTRIC AND ESOPHAGEAL VARICIES

D. PERIPHERAL EDEMA

E. HEPATIC ENCEPHALOPATHY

<<>>A, B, C

WHAT IS A LIFE THREATENING COMPLICATION OF CIRROHSIS?

A. PORTAL HYPERTENSION

WHAT ARE SOME LABS THAT WE MIGHT SEE IN A PATINET WITH CIRROHSIS? (SELECT ALL

THAT APPLY)

A. ANEMIA, THROMBOCYTOPENIA, LEUKOPENIA

B. ABNORMAL LIVER FUNCTIONS

C. DECREASED AMMONIA AND BILIRUBIN

D. DECREASED ALBUMIN AND POTASSIUM LEVELS

E. INCREASED INR

<<>>A, B, D,

E

WHAT CAN WE DO TO RELIEVE PRURITUS (ITCHING) IN CIRROHSIS PATIENTS?

A. LOTIONS, SOFT, OR OLD LINENS

B. TAKING HOT BATHS

C. APPLYING ICE TO THE SKIN

D. TAKING ANTIHISTAMINES

<<>>A

THE PATIENTS SKIN GETS ITCHY FROM INCREASE OF BILE SALTS IN THE BODY.

WE WANT TO DO TEMERPATURE CONTROL.

WHAT SHOULD A PATIENT DO IMMEDIATELY BEFORE A PARACENTESIS?

A. ADMINISTER THE PATIENTS DOSE OF GOLYTELY

B. STOP ALL THE PATIENTS LACTULOSE

C. HAVE THE PATIENT VOID

D. HAVE THE PATIENT BE ON BED REST 6 HOURS PRIOR

<<>>C

A PARACENTESIS IS A PROCEDURE IN WHICH THE PROVIDER REMOVES FLUID FROM THE

CAVITY.

IMMEDIATELY BEFORE WE HAVE THE PATIENT VOID. THE PROVIDER MIGHT

ACCIDENTALLY PUNCTURE THE BLADDER IF THE PATIENT DOESNT VOID PRIOR.

POST OP:

WE MONITOR THE PATIENT FOR HYPOVOLEMIA, BLEEDING, MONITOR BP AND HR. KEEP

THE PATIENT IN HIGH FOWLERS.

CARDIA CIRRHOSIS IS OFTEN CAUSED BY?

A. LONSTANDING SEVERE RIGHT SIDED HEART FAILURE

B. LONGSTANDING SEVERE LEFT SIDED HEART FAILURE

C. PATENT DUCTOUS ARTIOSUS THAT NEVER CLOSED AS A CHILD

D. CORONARY ARTERY DISEASE INCREASING PORTAL HYPERTENSION

<<>>A

CAUSES NECROSIS OF LIVER CELLS AND CIRHOSIS EVENTUALLY

FULMANINANT HEPATIC LIVER FAILURE IS DESCRIBED AS

A. GRADUAL ONSET AND LIFE THREATENING LIVER FAILURE

B. CHRONIC LIVER FAILURE SINCE CHILDHOOD

C. ACUTE LIVER FAILURE WITH RAPID ONSENT AND LIFE THREATENING

D. ACUTE LIVER FAILURE CAUSED BY GENETIC LINK

<<>>C

IT HAS A RAPID ONSET, AND IS LIFE THREATENING.

IT HAS ENCEPHALOPATHY.

CEREBAL EDEMA OCCURS LEADING TO ICP.

CHANGES IN MENTATION ARE THE FIRST SIGNS SEEN.