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A series of questions and verified correct answers related to neurological disorders, assessments, and gastrointestinal conditions. It covers topics such as sensory pathways, brain function, respiratory patterns, spinal shock, cerebrovascular accidents, multiple sclerosis, guillain-barre syndrome, neural tube defects, and various gastrointestinal disorders. The material is presented in a question-and-answer format, making it useful for exam preparation and review. It also includes key concepts related to liver and bowel diseases, such as portal hypertension, ascites, hepatic encephalopathy, and jaundice. Valuable for medical students and healthcare professionals seeking to reinforce their understanding of these critical topics. (400 characters)
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"What parts of the brain mediate the expression of affect, both emotional and behavioral
in PaCO2 levels" "Posthyperventilation apnea (PHVA) ceases and rhythmic breathing is resumed when levels of
decrescendo pattern of breathing, followed by a period of apnea" "Vomiting is associated with central nervous system (CNS) injuries that compress which of the
individual on the second day after hip replacement"
of autonomic functions such as gastrointestinal function"
the middle fossa through the tentorial notch into the posterior fossa"
return of spinal reflexes"
"A herniation of which disk will likely result in motor and sensory changes of the lateral lower
"A right h emisphere embolic CVA has resulted in left-sided paralysis and reduced sensation of
"Atrial fibrillation, rheumatic heart disease, and valvular prosthetics are risk factors for which
"Microinfarcts resulting in pure motor or pure sensory deficits are the result of which type of
"Which vascular malformation is characterized by arteries that feed directly into veins through
arteries"
demyelination, possible from an immunogenic virus" "What is the most common opportunistic infection associated with acquired immunodeficiency
of bright red blood in the stools, suggest a lower GI bleed usually in the rectum, sigmoid colon or descending colon"
esophagus, stomach or duodenum resulting in exposure of the tissue to gastric acid. Risk factors include smoking, advanced age, NSAID use, ETOH, chronic disease, acute pancreatitis, COPD, obesity, socioeconomic status, gastrinoma, and infection with Helicobacter pylori. S&S: Epigastric pain is worse with eating, melena or hematemesis"
patients. S&S: epigastric pain that is relieved by food. Patients may have melena(black and tarry stool) or hematemesis"
the large intestine. Risk factors include older age, genetic predisposition, obesity, smoking, diet, lack of exercise, ASA and other NSAIDS, altered DI microbiome and abnormal colonic peristalsis"
from the muscle layer of the intestine that protrude into the intestinal lumen most commonly in the sigmoid colon. Diverticulosis is the presence of diverticula in an asymptomatic person. Diverticulitis is an inflammation of diverticula and cause LLQ pain. Results in abscess formation, rupture and peritonitis"
S&S: periumbilical pain, RLQ pain, nvd, anorexia. Pain may initially be epigastric or periumbilical then settle in RLQ. Perforation, peritonitis and abscess formation are all potential complications"
adhesions, tumors, Crohn's disease, hernias and intussusception. SBO causes distention 2nd to impaired absorption and increased secretions which leads tofluid accumulation and gas proximal to the ileus. Distention in the intestines decreases their ability to absorb water and increases secretion of those things."
Lower in the GI tract -->metabolic acidsis. colicky abdominal pain, n/v, distention, if dehydrated then hypotension and tachycardia may occur, fever, leukocytosis and rebound tenderness if ischemia is present. Obstruction of pylorus results in proguse vomiting of clear gastric fluid. Partial SBO causes diarrhea. Complete SBO causes constipation and increased bowel sounds"
a tumor, but can also be diverticulitis, inflammatory bowel disease and volvulus. S&S: hypogastric pain, abdominal distention and vomiting which will occur late"
venous system caused by resistance to portal blood flow. Commonly caused by fibrosis, obstruction from cirrhosis, thrombosis, or narrowing of hepatic portal vein. Most common S&S is vomiting blood from bleeding esophageal varices."
complication of portal hypertension. Cirrhosis is most common cause. S&S: abdominal distension, increased abd girth, weight gain and in large volumes dyspnea, increased respiratory rate, peripheral edema, dilutional hyponatremia and may develop peritonitis."
causes the neurons to swell which leads to cerebral edema and IICP. Triggers for this include: ETOH abuse, infection, GI bleed, portal vein thrombosis, sedatives, volume depletion, constipation, electrolyte imbalances and diuretics. Asterixis AKA liver flap is most common sign."
skin and other tissues. Visible when bilirubin reaches 2.5-3mg/dL."
indirect(unconjugated) bilirubin typically from RBC hemolysis. Caused from ABO or RH incompatibility, sickle cell anemia, in newborns, and hepatocellular damage from hepatitis, cirrhosis or cancer. Total bili levels are elevated, direct bili is low and indirect bili is high"
fine but there is an obstruction of outward flow to the intestines. Most commonly caused by obstruction in the biliary tract. Hallmark sign is gray stools"
fibrotic scar tissue which is nonfunctioning. The injury results in cellular necrosis, inflammation and regeneration which leads to fibrosis. The fibrotic changes compress the blood vessels and develop portal hypertension."
albumin, ascites, decreased detox of medications, hormones and toxins, jaundice, esophagel and gastric varices, anemia, hematemesis/coffee ground emesis, black stools, splenomegaly,
positive and all other test are negative then person has acquired immunity from vaccine. Hep. B total core antibody positive and all other are negative, person had previous infection. Hep. B core antibody(IgM) is positive in acute infections."
hepatitis C infection"
include obesity, rapid weight loss, middle age, female gender, oral contraceptives, native american ancestry, ileal disease, low HDLs, malabsorption disorders and hypertriglyceridemia."
pass into the common bile duct and cause an obstruction which will cause painful spasms and contraction of the bile duct in the RUQ area called biliary colic. Pain is also felt in the back, right should or right scapula. N/V."
biliary tract. S&S: precipitated by a fatty meal, GERD, positive murphy's sign and rebound tenderness. Lab abnormalities include leukocytosis, increased alkaline phosphatase and direct bilirubin."
into the surrounding area. Enzymes begin the digestive process of the tissues they touch which may lead to hemorrhaging."
hyperlipidemia, smoking, some drugs, genetic factors, viral infections, autoimmune, ischemia, post ERCP, scorpion bite."
that radiates to the back and is worse when lying down, n/v and if hemorrhaging occurs then persons will have signs of hypovolemic shock, fever, hypocalcemia, jaundice and a lot of fluid loss"
consistent with acute pancreatitis, elevated lipase or findings consistent with acute pancreatitis on CT. Complications: ARDS, heart failure, renal failure, coagulopathies, sepsis, paralytic ileus and GI bleed"
and metaplastic changes in the esophagus. Do not show signs until later in the disease. 2 main symptoms are chest pain and dysphagia"
over 50.. Casues age, high fat, low fiber diets, smoking, obesity, family history, low levels of exercise, inflammatory bowel disease and gastrectomy. Commonly arise from polyps"
anemia, occult bleeding, obstruction, distention. Screening starts at age 50 and includes yearly test for occult blood, colonoscopy every 5-10 years, sigmoidoscopy every 5 years"
in males and african americans. Risk factors: ETOH use, family history, smoking, non O blood type, DM type 2, and chronic pancreatitis.K-ras mutation, a proto-oncogene is most common genetic alteration. S&S: jaundice, dull back pain, protein and fat malabsorption, lethary, weight loss, n/v, diabetes, changes in bowel patterns and pruritis"
development due to vit. B6, folic acid and B12 deficiencies, smoking, ETOH ingestion during pregnancy, steroid or statin use, maternal hyperhomocysteinemia, diabetes and genetic mutations. Cleft lips arise during the 4th week of gestation and cleft palate during the 3rd month"
food from stomach to deuodenum. most common cause of intestinal obstruction in infancy. Usually causes vomiting after eating. Increase gastrin secretion in 3rd trimester has been linked to cause this. Other causes include deficiency in nitric oxide synthase containingneurons, abnormal innervation of myenteric plexus and presence of infantile hypergastrinemia and exposure to macrolide antibiotics"
is greater than the 95th percentile for infants age in hours or bilirubin level greater than 20mg/dL. When bilirubin level reaches 2, jaundice is visible"
begins during the first week of life in healthy, full term infants. Caused by mild unconjugated hyperbilirubinemia. Subsides after a couple weeks"
levels>20 or indirect bilirubin>15. Risk factors include ABO or RH incompatibility, prematurity, exclusive breast feeding, maternal age greater than 25, male infant, delayed meconium passage and birth trauma. Most common cause is hemolytic disease of the newborn"
Medulla Oblongata" "Which area of the brain assumes responsibility for conscious and unconscious muscle synergy
modify spinal reflex arcs"
the cerebral venous sinuses"
"Which of the meninges closely adheres to the surface of the brain and spinal cord and follows
Vasoconstriction"
blood sugar levels"
Willis" "The nurse recognizes that a patient's diagnosis of a viral infection of the brain's meningeal
10/mm3"
"What is the most common infratentorial brain disease process that results in the direct
Cerebrovascular disease"
consciousness and the ability to react to exogenous stimuli"
Development of cerebral hypoxia" "The most critical aspect in correctly diagnosing a seizure disorder and establishing it's cause
"What type of seizure starts in the fingers and progressively spreads up the arm and extends to
Prefrontal"
verbal speech, but not comprehend language"
Tissue" "What type of cerebral edema occurs when permeability of the capillary endothelium increases
Fasciculations and muscle cramps" "The weakness resulting from the segmental paresis and paralysis characteristic of anterior
roots supply each muscle"
ganglia" "Clinical manifestations of Parkinson disease are caused by a deficit in which of the brain's
"Tremors at rest, rigidity, akinesia, and postural abnormalities are a resulte of the atrophy of
levels of consciousness"
"Which disorder has clinical manifestations that include decreased consciousness for up to 6
(Moderate) Cerebral Concussion"
Older adults" "The edema of the upper cervical cord after spinal cord injury is considered life threatening
impairment to the diaphragm"
the bladder" "What term is used to describe the complication that can result from a spinal cord injury above T6 that is producing paroxysmal hypertension, as well as piloerection and sweating above the
damage to the sympathetic nervous system" "Which clinial finding is considered a diagnostic indicator for an arteriovenous malformation
"Which cerebral vascular hemorrhage causes meningeal irritation, photophobia, and positive
cord"
"A blunt force injury to the forehead would result in a coup injury to which region of the brain?
"A blunt force injury to the forehead would result in a contrecoup injury to which rebion of the
thoracic-lumbar" "The most likelly rationale for body temperature fluctuations after cervical spinal cord injury is
resulting in disturbed thermal control." "A man who sustained a cervical spinal cord injury 2 days ago suddenly develops severe hypertension and bradycardia. He reports severe head pain and blurred vision. The most likely
autonomic hyperreflexia"
from the inferior mesenteric, gastric, and cystic veins. The hepatic portal vein receives deoxygenated blood from the inferior and superior mesenteric vein and splenic vein and delivers nutrients that have been absorbed from the intestinal system"
substance in the intestines. This pulls water by osmosis into the intestinal lumen and results in large volume diarrhea. This is how mag citrate, lactulose and miralax work. Causes include: excessive ingestion of nonabsorbable sugars, tube feedings, dumping syndrome, malabsorption, pancreatic enzyme deficiency, bile salt deficiency, small intestine bacterial overgrowth or celiac disease"
infectious causes such as rotavirus, bacterial enterotoxins, or c-diff."
resection of small intestine or surgical bypass of small intestine, IBS, diabetic neuropathy, hyperthyroidism, and laxative abuse. Fatty stools and bloating are common in malabsorption syndrome. Complications include: dehydration, electrolyte imbalance, metabolic acidosis, weight loss and malabsorption."
in persons 20-40y/o. Less common in people who smoke. Has periods of remission and exacerbations. Characterized by inflammation and ulcerations that remain superficial and in the small intestine."
uveitis, sclerosing cholangitis, erythema nodosum and pyoderma gangrenosum"
megacolon, colon perforation, and colorectal adenocarcinoma. Increased risk of VTE and microthrombi, and colon cancer"
portion of the GI tract but most often in the ileum and proximal colon. Affects persons in their 20-30s and of jewish decent. CARD15/NOD2 gene mutation commonly associated."
age less than 40, slight predominance in women and altered gut microbiome."
affected area(entire wall of intestine is affected) and the presence of skip lesions. Disease
progression may lead to abscess formation in GI tract. Possible causes include infectious agents, autoimmune, psychosomatic, impaired T-cell immunity"
malabsorption, malnutrition, weight loss, intestinal obstruction from chronic inflammation, fistulas and perforation of the intestine"